This 3D CT case-control study compares scapular morphology in normal shoulders, dynamic posterior instability, and static posterior humeral head subluxation. It asks whether these two clinically distinct conditions share a common bony phenotype beyond glenoid retroversion. Measurements covered glenoid version, inclination, and acromial tilt, coverage, and height.
When you see recurrent posterior instability in a young patient, look beyond the labrum. This study shows the underlying scapula carries increased glenoid retroversion plus a higher, flatter acromion with deficient posterolateral coverage.
That same bony phenotype appears in older patients with static posterior subluxation and eccentric arthritis. The shared shape suggests a common anatomical predisposition that may become symptomatic early as dynamic instability or late as static wear.
Practically, a soft-tissue repair alone may not address the bony deficiency driving posterior translation. Recognizing retroversion and posterior acromial deficiency on CT should raise consideration of bony procedures. Appraise this cautiously: the groups differed sharply in age and gender and were small, so causation versus adaptive remodeling remains unresolved. This remains debated.
This 3D CT case-control study compares scapular morphology in normal shoulders, dynamic posterior instability, and static posterior humeral head subluxation. It asks whether these two clinically distinct conditions share a common bony phenotype beyond glenoid retroversion. Measurements covered glenoid version, inclination, and acromial tilt, coverage, and height.
When you see recurrent posterior instability in a young patient, look beyond the labrum. This study shows the underlying scapula carries increased glenoid retroversion plus a higher, flatter acromion with deficient posterolateral coverage.
That same bony phenotype appears in older patients with static posterior subluxation and eccentric arthritis. The shared shape suggests a common anatomical predisposition that may become symptomatic early as dynamic instability or late as static wear.
Practically, a soft-tissue repair alone may not address the bony deficiency driving posterior translation. Recognizing retroversion and posterior acromial deficiency on CT should raise consideration of bony procedures. Appraise this cautiously: the groups differed sharply in age and gender and were small, so causation versus adaptive remodeling remains unresolved. This remains debated.