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Supracondylar Fractures of the Humerus in Children.

·J Am Acad Orthop Surg·1997·577 citations·Pediatrics
DOI·PubMed
SummaryAbstract on PubMed →

This 1997 review by Otsuka and Kasser covers the classification, radiographic evaluation, treatment algorithm, and complication management of supracondylar humerus fractures in children. It addresses the full spectrum from nondisplaced Type I injuries through completely displaced Type III fractures with neurovascular compromise. The central question is how to achieve anatomic reduction, stabilize the fracture, and manage the pulseless or nerve-injured limb.

Key Findings

  • Extension-type fractures account for 90%–98% of all supracondylar fractures, and displacement direction predicts which structure is at risk:
    –Posteromedial displacement → median nerve / anterior interosseous nerve
    –Posterolateral displacement → brachial artery
  • The Gartland classification links fracture pattern directly to treatment, eliminating guesswork:
    –Type I (nondisplaced) → cast or splint; no reduction needed
    –Type II (angulated, posterior cortex intact) → closed reduction; pin if unstable or swollen
    –Type III (completely displaced) → closed reduction + percutaneous pinning; casting alone is contraindicated
  • Nerve injury affects 5%–19% of all supracondylar fractures and is far more common in Type III injuries specifically:
    –Median nerve deficit — 52% of Type III cases
    –Radial nerve deficit — 28% of Type III cases
    –Nearly all are neurapraxias that resolve spontaneously; absence of recovery by 5 months warrants exploration and neurolysis
  • A pulseless hand after supracondylar fracture should trigger immediate closed reduction and pinning — not arteriography. In one series, pulse was restored in 13 of 17 pulseless patients after reduction alone, because the vessel is kinked rather than transected.
  • Cubitus varus develops in up to 58% of inadequately reduced Type III fractures and will not remodel — the distal physis contributes only 20% of humeral growth, making coronal plane malunion permanent. Corrective osteotomy carries a 33% complication rate, so anatomic primary reduction is the only reliable prevention.
Board PearlCubitus varus occurs in up to 58% of poorly reduced Type III supracondylar fractures and will not remodel — anatomic reduction at index surgery is the only prevention.

Clinical Relevance

Supracondylar humerus fracture is the most common elbow injury requiring hospitalization in children, and historically it carried high rates of malunion and neurovascular complications when managed with casting in hyperflexion or skeletal traction alone.

When you see a Type III supracondylar fracture, the management is closed reduction and percutaneous pinning — not casting. The direction of displacement tells you where to look first: posterolateral displacement means examine the brachial artery, posteromedial means test median nerve function carefully.

When the hand is pulseless after fracture, do not wait for arteriography. Reduce and pin immediately. Pulse returns in the majority of patients because the vessel is kinked, not transected. If pulse remains absent after reduction, then explore.

For pinning technique, placing the medial pin through a small incision with blunt dissection to the epicondyle protects the ulnar nerve and is the key step that prevents the 2%–3% iatrogenic nerve injury rate associated with percutaneous medial pin placement.

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|

Supracondylar Fractures of the Humerus in Children.

·J Am Acad Orthop Surg·1997·577 citations·Pediatrics
DOI·PubMed
SummaryAbstract on PubMed →

This 1997 review by Otsuka and Kasser covers the classification, radiographic evaluation, treatment algorithm, and complication management of supracondylar humerus fractures in children. It addresses the full spectrum from nondisplaced Type I injuries through completely displaced Type III fractures with neurovascular compromise. The central question is how to achieve anatomic reduction, stabilize the fracture, and manage the pulseless or nerve-injured limb.

Key Findings

  • Extension-type fractures account for 90%–98% of all supracondylar fractures, and displacement direction predicts which structure is at risk:
    –Posteromedial displacement → median nerve / anterior interosseous nerve
    –Posterolateral displacement → brachial artery
  • The Gartland classification links fracture pattern directly to treatment, eliminating guesswork:
    –Type I (nondisplaced) → cast or splint; no reduction needed
    –Type II (angulated, posterior cortex intact) → closed reduction; pin if unstable or swollen
    –Type III (completely displaced) → closed reduction + percutaneous pinning; casting alone is contraindicated
  • Nerve injury affects 5%–19% of all supracondylar fractures and is far more common in Type III injuries specifically:
    –Median nerve deficit — 52% of Type III cases
    –Radial nerve deficit — 28% of Type III cases
    –Nearly all are neurapraxias that resolve spontaneously; absence of recovery by 5 months warrants exploration and neurolysis
  • A pulseless hand after supracondylar fracture should trigger immediate closed reduction and pinning — not arteriography. In one series, pulse was restored in 13 of 17 pulseless patients after reduction alone, because the vessel is kinked rather than transected.
  • Cubitus varus develops in up to 58% of inadequately reduced Type III fractures and will not remodel — the distal physis contributes only 20% of humeral growth, making coronal plane malunion permanent. Corrective osteotomy carries a 33% complication rate, so anatomic primary reduction is the only reliable prevention.
Board PearlCubitus varus occurs in up to 58% of poorly reduced Type III supracondylar fractures and will not remodel — anatomic reduction at index surgery is the only prevention.

Clinical Relevance

Supracondylar humerus fracture is the most common elbow injury requiring hospitalization in children, and historically it carried high rates of malunion and neurovascular complications when managed with casting in hyperflexion or skeletal traction alone.

When you see a Type III supracondylar fracture, the management is closed reduction and percutaneous pinning — not casting. The direction of displacement tells you where to look first: posterolateral displacement means examine the brachial artery, posteromedial means test median nerve function carefully.

When the hand is pulseless after fracture, do not wait for arteriography. Reduce and pin immediately. Pulse returns in the majority of patients because the vessel is kinked, not transected. If pulse remains absent after reduction, then explore.

For pinning technique, placing the medial pin through a small incision with blunt dissection to the epicondyle protects the ulnar nerve and is the key step that prevents the 2%–3% iatrogenic nerve injury rate associated with percutaneous medial pin placement.

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